If you are an ECG instructor, you probably carefully choose ECGs to illustrate the topic you are teaching. One of the reasons for the existence of the ECG Guru website is our desire to provide lots of such illustrations for you to choose from.

Sometimes, though, an ECG does not clearly illustrate one specific dysrhythmia well, because the interpretation of the ECG depends on so many other factors.  In order to get it “right”, we would need to know information about the patient’s history, presentation, lab results, or previous ECGs. We might need to see the ECG done immediately before or after the one we are looking at.  Some ECG findings must ultimately be confirmed by an electrophysiology study before we can know for sure what is going on.

For those of us who are “ECG nerds”, it can be fun to debate our opinions and even more fun to hear from wiser, more advanced practitioners about their interpretations.

My belief, as a clinical instructor, is that we must teach strategies for treating the patient who has a “controversial” ECG that take into account the level of the practitioner, the care setting, and the patient’s hemodynamic status.  In some settings, it might be absolutely forbidden for a first-responder to cardiovert atrial fibrillation, for example.  But atrial fib is routinely cardioverted under controlled conditions in hospitals.  The general rule followed by emergency providers that “all wide-complex tachycardias are v tach until proven otherwise” has no doubt prevented deaths in situations where care providers did not agree on the origin of the tachycardia.

The ECG:    We do not have much patient information to go with this ECG, just that it is from a 71-year-old woman who developed severe hypotension and lost consciousness, but was revived with transcutaneous pacing.   Here is what we do know about this ECG:

·        There are regular P waves, at a rate of about 39 bpm (sinus bradycardia).

He presented to the hospital with a complaint of shortness of breath, and was determined to have an exacerbation of his COPD in the setting of a viral infection.  He was treated with breathing treatments and steroids, and was discharged home in improved condition.

The ECG   There is a sinus rhythm at 80 bpm.  The QRS complex is wide at .18 seconds (180 ms). The PR interval is slightly prolonged at .24 seconds (240 ms), which is first-degree AV block. There is right bundle branch block (QR in V1 and rS in Leads I and V6 with a wide QRS).  There is also a left posterior fascicular block (also called hemiblock), recognized by the right axis deviation (III is taller than aVF ).  This is bi-fascicular block, which can be chronic or acute.  There is very slight ST elevation with flattening in V1 and possibly V2.  In RBBB, “normal” T waves are directed opposite the terminal wave of the QRS.  V3 should, then, have inverted T waves. So, the upright T wave in this patient could be considered to be equivalent to an inversion in a normal ECG.  V4 through V6 have biphasic T waves, which is not normal for RBBB.  Because we do not have an old ECG, we don’t know which, if any, of these changes are chronic.  With this patient’s past medical history, it is possible for any of them to be pre-existing.

I will admit that, when I first saw this ECG, the V1 rhythm strip at the bottom looked as though there were regular P waves at a rate of about 220-240, with one buried in each QRS and one in each T wave. In scrutinizing all the other leads, I cannot find evidence for an underlying atrial tachycardia or atrial flutter, so it is probably a coincidence that the P and T “march out” with the assumption of a hidden P wave in the QRS.

 This ECG was obtained from an 84-year-old woman who was scheduled for surgery.  When the anesthesiologist did this ECG, the surgery was cancelled. It is a very good example of fascicular-level blocks. 

The underlying rhythm is a regular sinus rhythm at about 95 bpm.  There are some non-conducted P waves which are part of the sinus rhythm (not premature beats).  When the P waves DO conduct, the PR interval is steady at about .15 seconds (148 ms).

In addition, there is a LEFT BUNDLE BRANCH BLOCK.  The ECG criteria for LBBB are:  1) A supraventricular rhythm, 2) A wide QRS, and 3) A negative QRS in Lead V1 and a positive QRS in Leads I and V6.  The QRS duration in this ECG is 136 ms.

There are generally two fascicles (branches) in the left bundle branch, and one main fascicle in the right bundle branch.  So, a LBBB represents a “bi-fascicular block”.  That means that A-V conduction is proceeding down only one fascicle (the right bundle branch).  In that fascicle, there is an “intermittent” block.  When the RBB is not blocked, we see a QRS.  When it is blocked, we see none.  This is then termed an “intermittent tri-fascicular block” – otherwise known as SECOND-DEGREE AV BLOCK, TYPE II.  Type II blocks nearly always have a wide QRS due to the underlying bundle branch pathology.  You may see RBBB, LBBB, or RBBB with left anterior fascicular block (hemiblock).  Very rarely, the combination might include left posterior hemiblock.  The intermittent block in the “healthiest” fascicle(s) is what makes this a second-degree block, and not a complete heart block (third-degree AVB).

The clinical implications of this block are that the heart is operating on only one fascicle, and that fascicle is showing obvious signs of distress.  A third-degree AVB could be imminent.  In addition, LBBB causes a wide QRS, which decreases cardiac output.  Second-degree, Type II AVBs can result in very slow rates, and sometimes cause more hemodynamic instability that some third-degree AV blocks.

This patient was scheduled for pacemaker implantation instead of the originally-scheduled surgery.