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Dawn's picture

Anti-tachycardia Function of ICD

This ECG was donated to the ECG Guru by Brent Dubois, and was originally published on the FaceBook page, Paramedic Tips & Tricks.  We published it to this site three years ago, but believe it should be shown again, as it is somewhat rare to catch a good-quality 12-Lead ECG of an implanted cardioverter-defibrillator pacemaer using overdrive pacing to terminate a ventricular tachycardia.  Most of our examples have been rhythm strips.

In this strip, we see the patient in ventricular tachycardia (V tach) at a rate of about 190 / minute.  The ICD, in response to the fast rate, delivers a short burst of even faster paced beats.  The physological rule in the heart is, "the fastest pacemaker controls the heart".  Once the pacemaker has terminated the V tach, it paces at a much slower rate.  It is pacing the atria, and the conduction system is intact, allowing the impulse to travel normally through the ventricles.  If the sinus node is able to "outpace" the slower paced rhythm, the heart will resume a sinus rhythm.

This is called "overdrive pacing" and is done automatically by an ICD that is programmed to do so.  Overdrive pacing can also be accomplished by a temporary transvenous pacer or transcutaneous pacemaker.  

Dawn's picture

Right Ventricular Outflow Tract Tachycardia (RVOT)

This ECG was taken from a patient who was complaining of palpitations and tachycardia, but who was hemodynamically stable, with no history of heart disease.  It is an example of RIGHT VENTRICULAR OUTFLOW TRACT TACHYCARDIA, a type of idiopathic ventricular tachycardia.  The ECG signs of RVOT are:  wide QRS complex, left bundle branch block pattern (QRS negative in V1 and positive in Leads I and V6), heart rate over 100 bpm, rightward or inferior axis (LBBB usually has a normal to leftward axis), AV dissociation.

RVOT accounts for about 10% of all ventricular tachycardias, and 70% of idiopathic VT.  It is most often found in structurally normal hearts, but it may occur in patients with arrhythmogenic right ventricular dysplasia.  For more on RVOT, read Life In the Fast Lane.

RVOT tachycardia sometimes converts with adenosine.  The patient in this example converted after being administered amiodarone.

Dawn's picture

Wide Complex Tachycardia

Wide-QRS rhythms can be difficult to diagnose from the ECG alone.  This difficulty is compounded when the rate is fast, as it can be hard to determine if P waves are present before the QRSs, or dissociated, or absent.

This ECG and rhythm strip were donated to the ECG Guru by Ryan Cihowiak.  We don't have clinical information on the patient, unfortunately.  It is a great example, however, of how difficult WCT can be to diagnose.

In the 12-Lead ECG, we see wide QRS complexes that are regular at a rate of 131 / minute. There are no obvious P waves before the QRS complexes, and no obvious distortion of the T waves, which would suggest a "hidden" P wave.  Unfortunately, there is significant artifact, which makes searching for P waves difficult.  The pattern overall suggests left bundle branch block, with the negative QRS in Lead V1 and positive QRS complexes in Leads I and V6.  However, one requirement for the diagnosis of LBBB is a supraventricular rhythm, and P waves are the best indicator of that.  An irregularly-irregular rhythm, indicating atrial fib, would also have made LBBB more likely.  In typical LBBB, the frontal plane axis is usually left-normal or left.  In this ECG, Lead III is taller than Lead I, putting the axis within normal range, but slightly rightward.

The rhythm strip uncovers something else.  Possible P waves are seen in some of the ST segments (arrows).  Are these dissociated?  Do they represent a first-degree AV block?  Are they actually artifact?  If this is a supraventricular rhythm, there is LBBB.   Then, notice beats #7,8,9.  If this rhythm is supraventricular (with LBBB), those must be a salvo of V Tach.  But, one of the possible P waves occurs in front of beat #7.

Another possibility is Right Ventricular Outflow Tract Tachycardia.  RVOT is a type of V Tach that typically has a LBBB pattern, with a slightly rightward axis.  If this is the case, beats #7,8,9 are probably "capture" beats or "fusion" beats.  Capture and fusion beats "prove" that the underlying tachycardia is ventricular, since, by definition, capture and fusion represent a return to supraventricular control of the rhythm.

Dawn's picture

ECG Basics: Torsades Cardioverted

These two strips are from one patient who was electrically cardioverted twice in a few minutes.  The original reason for the cardioversion was Torsades de Pointes, a type of polymorphic ventricular tachycardia associated with a long QT interval.  For more information about TDP, go to this LINK.  It is a bit difficult to comment on the patient's post-cardioversion rhythm, because so little of it is shown.  It appears to be sinus, with a wide QRS.  The QT interval appears slightly prolonged at .44 sec, but it is not known what the QT interval is corrected to a rate of 60/min.  TDP is often seen with QT intervals greater than 600 ms (.6 seconds).  Also THESE STRIPS ARE NOT SIMULTANEOUS, they were taken two minutes apart.  In the first one, the P waves and T waves look so much alike, they could all be P waves.  They do not "march out".  It is necessary to get a long strip, preferably in multiple leads, and a 12-Lead ECG, to properly evaluate the rhythm post-cardioversion. 

Dawn's picture

Teaching Series 1113: ECG 5 of 6 - Acute Anterior Wall M.I.

Continuing our series from the patient with acute AWMI, donated by Jenda Enis Štros, a new dysrhythmia has appeared.  The patient was taken to the cath lab, and a thrombus was removed from the stent.   We now see a wide-complex tachycardia.  The morphology (shape and configuration) of the QRS has changed considerably from the previous ECGs, so we know this is idioventricular in origin.  There are many other clues, but  some major ones are:  the QRS is very wide, there is no associated P wave, all the precordial leads except V1 are negative (precordial concordance), V6 is negative, and the axis is away from II, III, and aVF and toward aVR (aVR is upright).  

Because the rate is about 125/min., this could be called ventricular tachycardia (V Tach) or accelerated idioventricular rhythm (AIVR).  The important fact here is that this rhythm was transitory and the patient remained stable.  Reperfusion dysrhythmias are not uncommon, and the patient is treated as indicated by the clinical condition. 

Here are links to all six ECGs in this series:

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-1-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-2-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-3-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-4-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-5-6-acute-anterior-wall-mi 

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-6-6-acute-anterior-wall-mi

 

   

Dawn's picture

Teaching Tip: 12 Leads are Better Than One (Or Three)

Years ago, I was tasked with introducing 12-lead ECG interpretation to firefighter/paramedics who had been using ECG for rhythm monitoring for years.  Some were eager to add to their skills, others - not so much.  The feeling was, we have been doing just fine as we are.  When finally convinced that they could interpret STEMI with a 12-lead, many were content to use the 12-lead ECG only for that.  

To illustrate to students the great value of multi-lead assessment, I devised a little "quiz".  I showed the students ten to twelve short rhythm strips, like you see here.  All were cropped from 12-lead ECGs.  I asked my class to interpret the strips as they would if they were taking an ACLS class.  Usually, all did fine, or so they thought.  When shown the 12-lead ECGs the strips were taken from, EVERY student changed his or her mind on EVERY ECG.  The lesson is:  sometimes what we are looking for shows up in some leads and not others.  You can find this illustrated hundreds of times just in the ECG archives on this site.  I will supply some ECGs here on this page over the next few weeks that you could use to show your own students the value of "multi-lead assessment".  

What started as a hard-sell turned out to be a fun exercise.

The ECG shown here is of a patient in V Tach.  There are several strong signs that this is V Tach, including the wide QRS complexes, lack of associated P waves, "backward" axis, also called extreme right axis deviation (Leads II, III, and aVF are all negative and aVR is positive), and V6 is negative.  For more review of the differential diagnosis of wide-complex tachycardias, go to our Ask the Expert answer from Jason Roediger.  This LINK willl take you to Dr. Grauer's informative webpage where he offers a step-by-step guide to differentiating the WCTs.

 The focus of THIS lesson is that, while the patient is in V Tach, and it is in every lead, the tell-tale signs are harder to see in some leads than others.  Remember to show your  Remember to share with your students that the channels of the ECG (in this case three) are run simultaneously, so that the same heartbeat is seen several times - once for each channel.

Dawn's picture

ECG Basics: Ventricular Tachycardia

An example of ventricular tachycardia in Lead II.  This patient's rate is about 190/min.  V Tach will have the following criteria:  Rate greater than 100/min, QRS duration greater than .12 sec. (120 ms), and no P wave associated with the QRS.

It can be difficult to distinguish V Tach from other wide-complex tachycardias without a 12-lead ECG, but all wide-complex tachycardias should be treated as V Tach until proven otherwise, as V Tach is a potentially lethal dysrhythmia.  V Tach can cause a severe reduction in cardiac output which can lead to V Fib and death.

Dawn's picture

ECG Basics: Torsades de Pointes

Torsades de pointes, or polymorphic ventricular tachycardia, is a ventricular tachycardia precipitated by and associated with long QT Syndrome.  Long QT Syndrome can be congenital or acquired.  Torsades is life-threatening, and can be made worse by many drugs, including some of the drugs used to treat VT.  The rate is usually 150 - 250 / min. and the appearance is of a wide-complex tachycardia with QRS morphology changes.  In some leads, it will appear as if it is "twisting" around the isoelectric line, giving it the French name, Torsades de pointes, a ballet term meaning twisting of the points.  For a thorough discussion of Torsades, check this LINK.

Dawn's picture

Ventricular Tachycardia

This is a good example of ventricular tachycardia with PRECORDIAL CONCORDANCE.  The QRS complexes in the chest, or precordial, leads all point downward.  When the precordial leads are all  negative or all positive in a wide-complex tachycardia, there is virtually a 100% chance that the WCT is ventricular tachycardia.  This ECG shows many characteristics of VT, including the extreme "backwards" axis:  aVR is positive and II, III, and aVF are negative.  Lead I is almost equiphasic.  Also, the lack of a clear BBB pattern and a negative V6 are strongly suggestive of VT.  REMEMBER:  In the treatment of wide-complex tachycardia, the rhythm should be considered VT unless proven otherwise.  This is especially true in unstable patients, patients over 50 years old, and patients with known heart disease.

INSTRUCTORS' NOTE:  We purposely left the machine interpretation on this week's ECG of the Week.  How many errors did the machine make?  This might be a good teaching point for students of all levels.

 

jer5150's picture

Jason's Blog: ECG Challenge of the Bi-Week for Nov. 18th - Dec. 1st.

Patient's clinical data:  64-year-old white man.

What is the rhythm seen in this 12-lead ECG?

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